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Mechanisms and risk factors for perinatal allergic disease
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B cells spatial organization defines their phenotype and function in cancer “Tell me with whom you consort, and I will tell you who you are” ― Goethe
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Epithelial-immune interactions govern type 2 immunity at barrier surfaces
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Metabolic adaptations of ILC2 and Th2 cells in type 2 immunity
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Tissue-resident memory cells in antitumoral immunity and cancer immunotherapy
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Metabolic requirements of type 2 lymphocytes in allergic disease
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The immunometabolic roots of aging
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Host-encoded antivirulence defenses: host physiologies teach pathogens to play nice
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The path ahead for understanding Toll-like receptor-driven systemic autoimmunity
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Metabolism and macrophages in the tumor microenvironment
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Carbohydrate metabolism in supporting and regulating neutrophil effector functions
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Molecular mechanisms regulating T helper 2 cell differentiation and function
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Role of allergen immunotherapy and biologics in allergic diseases
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Fuelling B cells: dynamic regulation of B cell metabolism
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Immunometabolic regulation of germinal centers and its implications for aging
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Regulation of inflammation by Interleukin-10 in the intestinal and respiratory mucosa
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The path ahead for understanding Toll-like receptors–driven systemic autoimmunity
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Eating for immunity: how diet shapes our defenses
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Inflammasomes and their role in PANoptosomes
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B cell memory of Immunoglobulin E (IgE) antibody responses in allergy
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The role of intestinal bacteria in promoting tolerance to food
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Epithelial sensing in allergic disease
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Multilevel approaches to immunization equity
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Epithelial barrier dysfunction, type 2 immune response, and the development of chronic inflammatory diseases
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Metabolic footprint and logic through the T cell life cycle
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Rethinking Toll-like receptor signalling
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Microbiome modulation of antigen presentation in tolerance and inflammation
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Powerful microscopy technologies decode spatially organized cellular networks that drive response to immunotherapy in humans
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T follicular helper cells in food allergy
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Linking signal input, cell state, and spatial context to inflammatory responses
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