Normally, the components of the vitreomacular interface, like collagen, chondroitin, laminin, and fibronectin, help in the binding of the posterior vitreous to the retina (internal limiting membrane) [1]. In the eyes with PDR, the hyperglycemia-induced cascade of complement-induced mechanisms leading to the uneven separation of the vitreous from the retina can result in localized TRD, which is the probable reason for the unusual morphology of TRD in this case [2]. Ultra-wide-field imaging of the fundus helps in the documentation and surgical planning of these eyes.