Trigger finger is a common hand disorder that causes pain, snapping and disability of the finger or thumb. It is caused by increased synovium volume around the finger flexor tendons, creating a mechanical obstacle to tendon gliding under the A1 pulley [1]. The lifetime incidence of this condition in the general population is 2–3%, with higher prevalence in females and 15% incidence in manual workers [2]. Several systemic endocrine and inflammatory conditions are risk factors for onset or and aggravation. Diabetes mellitus significantly increases the risk of developing trigger finger, with 5–20% prevalence in the diabetic population [3].

In early stages, conservative treatment by splinting to block the proximal interphalangeal (PIP) joint, oral medication, physical therapy and/or corticosteroid injection, can be applied [4], [5], [6]. However, many patients do not respond to conservative treatment, and surgery is inevitable. Surgical treatment typically uses an open technique. The success rate of open trigger finger release is 90–100%, while the risk of complications such as complex regional pain syndrome, infection, nerve injury, stiffness, scar pain, incomplete release and flexion deformity is 5–12% [2], [7].

Recently, endoscopic trigger finger release was developed to minimize recovery time, allow quicker return to activities, and decrease the rate of complications [8], [9], [10].

Despite positive expectations, outcomes have rarely been reported. The present study reported outcomes of 2154 endoscopic trigger finger release procedures performed in our institution.