1. Savarino, E, de Bortoli, N, De Cassan, C, et al. The natural history of gastro-esophageal reflux disease: a comprehensive review. Dis Esophagus 2017; 30: 1–9.
Google Scholar | Medline2. El-Serag, HB, Sweet, S, Winchester, CC, et al. Update on the epidemiology of gastro-oesophageal reflux disease: a systematic review. Gut 2014; 63: 871–880.
Google Scholar | Crossref | Medline | ISI3. Fujiwara, Y. [Recent epidemiology of GERD in the Japanese population]. Nihon Shokakibyo Gakkai Zasshi 2017; 114: 1781–1789.
Google Scholar | Medline4. Fujiwara, Y, Arakawa, T. Epidemiology and clinical characteristics of GERD in the Japanese population. J Gastroenterol 2009; 44: 518–534.
Google Scholar | Crossref | Medline | ISI5. Asaka, M, Kato, M, Kudo, M, et al. Atrophic changes of gastric mucosa are caused by Helicobacter pylori infection rather than aging: studies in asymptomatic Japanese adults. Helicobacter 1996; 1: 52–56.
Google Scholar | Crossref | Medline6. Hojo, M, Miwa, H, Ohkusa, T, et al. Alteration of histological gastritis after cure of Helicobacter pylori infection. Aliment Pharmacol Ther 2002; 16: 1923–1932.
Google Scholar | Crossref | Medline7. Calam, J. Helicobacter pylori modulation of gastric acid. Yale J Biol Med 1999; 72: 195–202.
Google Scholar | Medline8. Beales, IL, Calam, J. Interleukin 1 beta and tumour necrosis factor alpha inhibit acid secretion in cultured rabbit parietal cells by multiple pathways. Gut 1998; 42: 227–234.
Google Scholar | Crossref | Medline | ISI9. El-Omar, EM, Oien, K, El-Nujumi, A, et al. Helicobacter pylori infection and chronic gastric acid hyposecretion. Gastroenterology 1997; 113: 15–24.
Google Scholar | Crossref | Medline | ISI10. Furuta, T, Baba, S, Takashima, M, et al. Effect of Helicobacter pylori infection on gastric juice pH. Scand J Gastroenterol 1998; 33: 357–363.
Google Scholar | Crossref | Medline11. Kinoshita, Y, Kawanami, C, Kishi, K, et al. Helicobacter pylori independent chronological change in gastric acid secretion in the Japanese. Gut 1997; 41: 452–458.
Google Scholar | Crossref | Medline12. Delaney, B, McColl, K. Review article: Helicobacter pylori and gastro-oesophageal reflux disease. Aliment Pharmacol Ther 2005; 22(Suppl. 1): 32–40.
Google Scholar | Crossref | Medline13. Wu, JC. Does Helicobacter pylori infection protect against esophageal diseases in Asia? Indian J Gastroenterol 2011; 30: 149–153.
Google Scholar | Crossref | Medline14. Gunji, T, Sato, H, Iijima, K, et al. Risk factors for erosive esophagitis: a cross-sectional study of a large number of Japanese males. J Gastroenterol 2011; 46: 448–455.
Google Scholar | Crossref | Medline15. Iijima, K, Ohara, S, Sekine, H, et al. Changes in gastric acid secretion assayed by endoscopic gastrin test before and after Helicobacter pylori eradication. Gut 2000; 46: 20–26.
Google Scholar | Crossref | Medline16. Tucci, A, Biasco, G, Paparo, GF. Effect of eradication of Helicobacter pylori in patients with fundic atrophic gastritis. N Engl J Med 1997; 336: 957–958.
Google Scholar | Crossref | Medline17. el-Omar, EM, Penman, ID, Ardill, JE, et al. Helicobacter pylori infection and abnormalities of acid secretion in patients with duodenal ulcer disease. Gastroenterology 1995; 109: 681–691.
Google Scholar | Crossref | Medline | ISI18. Haruma, K, Mihara, M, Okamoto, E, et al. Eradication of Helicobacter pylori increases gastric acidity in patients with atrophic gastritis of the corpus-evaluation of 24-h pH monitoring. Aliment Pharmacol Ther 1999; 13: 155–162.
Google Scholar | Crossref | Medline | ISI19. Labenz, J, Blum, AL, Bayerdörffer, E, et al. Curing Helicobacter pylori infection in patients with duodenal ulcer may provoke reflux esophagitis. Gastroenterology 1997; 112: 1442–1447.
Google Scholar | Crossref | Medline | ISI20. Sugimoto, M, Murata, M, Mizuno, H, et al. Endoscopic reflux esophagitis and reflux-related symptoms after Helicobacter pylori eradication therapy: meta-analysis. J Clin Med 2020; 9: 3007.
Google Scholar | Crossref21. Take, S, Mizuno, M, Ishiki, K, et al. Helicobacter pylori eradication may induce de novo, but transient and mild, reflux esophagitis: prospective endoscopic evaluation. J Gastroenterol Hepatol 2009; 24: 107–113.
Google Scholar | Crossref | Medline22. Hamada, H, Haruma, K, Mihara, M, et al. High incidence of reflux oesophagitis after eradication therapy for Helicobacter pylori: impacts of hiatal hernia and corpus gastritis. Aliment Pharmacol Ther 2000; 14: 729–735.
Google Scholar | Crossref | Medline | ISI23. Satoh, K, Yoshino, J, Akamatsu, T, et al. Evidence-based clinical practice guidelines for peptic ulcer disease 2015. J Gastroenterol 2016; 51: 177–194.
Google Scholar | Crossref | Medline24. von Elm, E, Altman, DG, Egger, M, et al. The strengthening the reporting of observational studies in epidemiology (STROBE) statement: guidelines for reporting observational studies. Ann Intern Med 2007; 147: 573–577.
Google Scholar | Crossref | Medline | ISI25. Lundell, LR, Dent, J, Bennett, JR, et al. Endoscopic assessment of oesophagitis: clinical and functional correlates and further validation of the Los Angeles classification. Gut 1999; 45: 172–180.
Google Scholar | Crossref | Medline | ISI26. Toyoshima, O, Nishizawa, T, Koike, K. Endoscopic Kyoto classification of Helicobacter pylori infection and gastric cancer risk diagnosis. World J Gastroenterol 2020; 26: 466–477.
Google Scholar | Crossref | Medline27. Kono, S, Gotoda, T, Yoshida, S, et al. Can endoscopic atrophy predict histological atrophy? Historical study in United Kingdom and Japan. World J Gastroenterol 2015; 21: 13113–13123.
Google Scholar | Crossref | Medline28. Kimura, KTT . An endoscopic recognition of the atrophic border and its significance in chronic gastritis. Endoscopy 1969; 1: 87–97.
Google Scholar | Crossref29. Iwakiri, K, Kinoshita, Y, Habu, Y, et al. Evidence-based clinical practice guidelines for gastroesophageal reflux disease 2015. J Gastroenterol 2016; 51: 751–767.
Google Scholar | Crossref | Medline30. Kodama, M, Murakami, K, Okimoto, T, et al. Ten-year prospective follow-up of histological changes at five points on the gastric mucosa as recommended by the updated Sydney system after Helicobacter pylori eradication. J Gastroenterol 2012; 47: 394–403.
Google Scholar | Crossref | Medline31. Ohkusa, T, Fujiki, K, Takashimizu, I, et al. Improvement in atrophic gastritis and intestinal metaplasia in patients in whom Helicobacter pylori was eradicated. Ann Intern Med 2001; 134: 380–386.
Google Scholar | Crossref | Medline | ISI32. Nakajima, S, Hattori, T. Active and inactive gastroesophageal reflux diseases related to Helicobacter pylori therapy. Helicobacter 2003; 8: 279–293.
Google Scholar | Crossref | Medline33. Yoshimura, M, Nagahara, A, Ohtaka, K, et al. Presence of vertebral fractures is highly associated with hiatal hernia and reflux esophagitis in Japanese elderly people. Intern Med 2008; 47: 1451–1455.
Google Scholar | Crossref | Medline34. Lender, N, Talley, NJ, Enck, P, et al. Review article: associations between Helicobacter pyloriand obesity – an ecological study. Aliment Pharmacol Ther 2014; 40: 24–31.
Google Scholar | Crossref | Medline | ISI35. Azuma, T, Suto, H, Ito, Y, et al. Eradication of Helicobacter pylori infection induces an increase in body mass index. Aliment Pharmacol Ther 2002; 16(Suppl. 2): 240–244.
Google Scholar | Crossref | Medline36. Takao, T, Ishikawa, T, Ando, T, et al. Multifaceted assessment of chronic gastritis: a study of correlations between serological, endoscopic, and histological diagnostics. Gastroenterol Res Pract 2011; 2011: 631461.
Google Scholar | Crossref | Medline