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An error was made in generating Figure S10 of this article [1]: the GADPH control blot for the experiment shown in Figure 7B was duplicated in the GADPH panel of Figure S10B in error and mislabelled. An updated version of Figure S10 is provided with this Correction. The Id1 and GAPDH blots in panel B have been replaced with the correct data from a different independent replicate carried out as part of the original experiments, and correctly labelled as Id1 and Actin; these errors do not affect the quantitative data reported in panel C. Underlying data supporting the updated figure are in S1, S2, and S3 Files. The authors confirm that Figure 7B in the published article showed the correct data.
The authors apologise for the errors in the published article.
Raw data supporting other results in the published article are available from the corresponding author.
Supporting informationFigure S10. Overexpression of Id1 only did not induce MM cell transformation.(A) Overexpression of Id1 did not support anchorage-independent growth of MM cells in soft agar assay (B) Id1 expression was detected in MM-Id1 clones and MM cells by immunoblotting. (C) The relative expression of Id1 in the representative blot in panel B was quantified by densitometric analysis, n = 1.
https://doi.org/10.1371/journal.ppat.1010390.s001
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Reference1. Liang D, Hu H, Li S, Dong J, Wang X, Wang Y, et al. (2014) Oncogenic Herpesvirus KSHV Hijacks BMP-Smad1-Id Signaling to Promote Tumorigenesis. PLoS Pathog 10(7): e1004253. https://doi.org/10.1371/journal.ppat.1004253 pmid:25010525Citation: Liang D, Hu H, Li S, Dong J, Wang X, Wang Y, et al. (2022) Correction: Oncogenic Herpesvirus KSHV Hijacks BMP-Smad1-Id Signaling to Promote Tumorigenesis. PLoS Pathog 18(3): e1010390. https://doi.org/10.1371/journal.ppat.1010390
Published: March 14, 2022
Copyright: © 2022 Liang et al. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
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