Extraglomerular immune complex deposition in lupus nephritis

1. Schwartz, N, Goilav, B, Putterman, C. The pathogenesis, diagnosis and treatment of lupus nephritis. Curr Opin Rheumatol 2014; 26: 502–509.
Google Scholar | Crossref | Medline2. Satoskar, A, Brodsky, S, Nadasdy, G, et al. Discrepancies in glomerular and tubulointerstitial/vascular immune complex IgG subclasses in lupus nephritis. Lupus 2011; 20: 1396–1403.
Google Scholar | SAGE Journals | ISI3. Chang, A, Henderson, SG, Brandt, D, et al. In situ B cell-mediated immune responses and tubulointerstitial inflammation in human lupus nephritis. J Immunol 2011; 186: 1849–1860.
Google Scholar | Crossref | Medline | ISI4. Wang, H, Xu, J, Zhang, X, et al. Tubular basement membrane immune complex deposition is associated with activity and progression of lupus nephritis: a large multicenter Chinese study. Lupus 2018; 27: 545–555.
Google Scholar | SAGE Journals | ISI5. Hsieh, C, Chang, A, Brandt, D, et al. Predicting outcomes of lupus nephritis with tubulointerstitial inflammation and scarring. Arthritis Care Res 2011; 63: 865–874.
Google Scholar | Crossref | Medline | ISI6. Hill, GS, Delahousse, M, Nochy, D, et al. A new morphologic index for the evaluation of renal biopsies in lupus nephritis. Kidney Int 2000; 58: 1160–1173.
Google Scholar | Crossref | Medline | ISI7. Yu, F, Wu, LH, Tan, Y, et al. Tubulointerstitial lesions of patients with lupus nephritis classified by the 2003 international society of nephrology and renal pathology society system. Kidney Int 2010; 77: 820–829.
Google Scholar | Crossref | Medline | ISI8. Londoño Jimenez, A, Mowrey, WB, Putterman, C, et al. Brief report: tubulointerstitial damage in lupus nephritis: a comparison of the factors associated with tubulointerstitial inflammation and renal scarring. Arthritis Rheumatol 2018; 70: 1801–1806.
Google Scholar | Crossref | Medline9. Lan-ting, H, You-ming, C, Li-xin, W, et al. Clinicopathological factors for tubulointerstitial injury in lupus nephritis. Clin Rheumatol 2020; 39: 1617–1626. DOI: 10.1007/s10067-019-04909-3.
Google Scholar | Crossref | Medline10. Park, MH, D’Agati, V, Appel, GB, et al. Tubulointerstitial disease in lupus nephritis: relationship to immune deposits, interstitial inflammation, glomerular changes, renal function, and prognosis. Nephron 1986; 44: 309–319.
Google Scholar | Crossref | Medline | ISI11. Park, MH, D’Agati, V, Appel, GB, et al. Tubulointerstitial disease in lupus nephritis: relationship to immune deposits, interstitial inflammation, glomerular changes, renal function, and prognosis. Nephron 1986; 44: 309–319.
Google Scholar | Crossref | Medline | ISI12. Hsieh, C, Chang, A, Brandt, D, et al. Predicting outcomes of lupus nephritis with tubulointerstitial inflammation and scarring. Arthritis Care Res 2011; 63: 865–874.
Google Scholar | Crossref | Medline | ISI13. Mori, Y, Kishimoto, N, Yamahara, H, et al. Predominant tubulointerstitial nephritis in a patient with systemic lupus nephritis. Clin Exp Nephrol 2005; 9: 79–84.
Google Scholar | Crossref | Medline14. Omokawa, A, Wakui, H, Okuyama, S, et al. Predominant tubulointerstitial nephritis in a patient with systemic lupus erythematosus: phenotype of infiltrating cells. Clin Nephrol 2008; 69: 436–444.
Google Scholar | Crossref | Medline15. Ravindran, A, Casal Moura, M, Fervenza, FC, et al. In patients with membranous lupus nephritis, exostosin-positivity and exostosin-negativity represent two different phenotypes. J Am Soc Nephrol 2021; 32: 695–706.
Google Scholar | Crossref | Medline16. Sethi, S, Debiec, H, Madden, B, et al. Semaphorin 3B-associated membranous nephropathy is a distinct type of disease predominantly present in pediatric patients. Kidney Int 2020; 98: 1253–1264.
Google Scholar | Crossref | Medline17. Kinloch, AJ, Cascino, MD, Dai, J, et al. Anti-vimentin antibodies: a unique antibody class associated with therapy-resistant lupus nephritis. Lupus 2020; 29: 569–577.
Google Scholar | SAGE Journals | ISI

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